Cerebral 5-HT2A receptor binding is increased in patients with Tourette's syndrome

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Cerebral 5-HT2A receptor binding is increased in patients with Tourette's syndrome.

Experimental and clinical data have suggested that abnormalities in the serotonergic neurotransmissions in frontal-subcortical circuits are involved in Tourette's syndrome. To test the hypothesis that the brain's 5-HT2A receptor binding is increased in patients with Tourette's syndrome, PET imaging was performed. Twenty adults with Tourette's syndrome and 20 healthy control subjects were invest...

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UNLABELLED Indirect estimations of brain neurotransmitters in patients with anorexia nervosa (AN) and low weight have demonstrated a reduction in brain serotonin (5-HT) turnover in general and led to hypotheses about dysfunction in the 5-HT(2a) receptor system. It was our aim to investigate the central 5-HT(2a) receptor binding index using SPECT brain imaging. METHODS The 5-HT(2a) receptors o...

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Patients with obsessive-compulsive disorder have increased 5-HT2A receptor binding in the caudate nuclei.

The pharmacological efficacy of serotonergic-acting drugs suggest that patients with obsessive-compulsive disorder (OCD) may have alterations in their cerebral serotonergic (5-HT) receptor system, and previous neuroimaging studies of OCD patients have shown abnormalities in several fronto-subcortical regions. In this study we investigated cerebral 5-HT(2A) receptor binding in 15 untreated OCD p...

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HT2A Receptor Binding is Increased in Borderline ersonality Disorder

ackground: Postmortem studies in suicide victims demonstrate an increase in the number of post-synaptic 5-HT2A receptor binding sites in entral lateral and orbital frontal cortex. Diminished metabolic responses to serotonergic activation are noted in these areas in impulsive subjects ith borderline personality disorder (BPD), a group at high risk for suicidal behaviors. We examined 5HT2A recept...

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Reduced 5-HT2A receptor binding after recovery from anorexia nervosa.

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ژورنال

عنوان ژورنال: The International Journal of Neuropsychopharmacology

سال: 2006

ISSN: 1461-1457,1469-5111

DOI: 10.1017/s1461145706006559